Abs ac s
S218
In e na ional Jou nal o Obesi y
T5:PS.157
Obesi y and as hma in schoolchild en
Pad ão, P1, Mo ei a, P1,2, Valen e, H¹, Co dei o, T¹, Bessa, M¹, Lopes, C3, Mo ei a, A4
¹ Facul y o Nu i ion and Food Sciences, Uni e si y o Po o, Po o, Po ugal
2Resea ch Cen e in Physical Ac i i y, Heal h and Leisu e, Uni e si y o Po o,
Po o, Po ugal
3 Depa men o Hygiene and Epidemiology, Uni e si y o Po o Medical School,
Po o, Po ugal
4 Depa men o Immunology, Uni e si y o Po o Medical School, Po o, Po ugal
and Depa men o Immunoalle gology, Uni e si y Hospi al o São João, Po o,
Po ugal
Backg ound: As hma and obesi y a e majo public heal h p oblems in childhood,
bu con o e sy emains ega ding he mechanisms unde lying his ela ionship. The
aim o his s udy was o e alua e he associa ion be ween as hma and obesi y isk in
schoolchild en.
Pa icipan s and me hods: The s udy was pe o med using a con enience sample
o 1962 Po uguese school child en (49.8% gi ls), 5-10-yea -old. Heigh and weigh
we e measu ed acco ding o in e na ional s anda ds, and body mass index (BMI) was
calcula ed. The de ini ion o obesi y was based on a e age cen iles acco ding o he
In e na ional Obesi y Task Fo ce cu -o s. Child en’s pa en s comple ed a sel -
adminis e ed ques ionnai e, which p o ided in o ma ion on gene al amily
backg ound cha ac e is ics, child en’s die a y in ake (using a semi-quan i a i e ood
equency ques ionnai e) and as hma (based on he ollowing ques ion: “Ha e you
e e been old by any doc o ha you son had as hma?”. Uncondi ional logis ic
eg ession models we e i ed o es ima e he magni ude o he associa ion be ween
as hma and obesi y in child en, adjus ing o con ounde s (age and ene gy in ake).
Resul s: The p e alence o obesi y was 11.0% in gi ls and 14.0% in boys and he
p e alence o as hma was 7.3% in gi ls and 11.0% in boys. Gi ls epo ing as hma
p esen ed a wo- old isk o being obese (OR = 2.07, 95% CI 1.06-4.05, p end =
0.05), e en a e u he adjus men o con ounde s (OR = 2.16, 95% CI 1.10-4.24,
p end = 0.05). In boys, no associa ion was ound be ween as hma and obesi y.
Conclusion: As hma was posi i ely associa ed wi h obesi y in gi ls.
T5:PS.158
Obesi y emains unde diagnosed in english hospi al in-pa ien s
Sha ma, V1,Ha ge , S2, B ai hwai e, A 2, Fine , N 1,2
1Lu on and Duns able Hospi al NHS Founda ion T us , Lu on, UK
2Uni e si y o Camb idge School o Clinical Medicine, Camb idge, UK
O ganised medical managemen o obesi y in England emains poo ly de eloped; hospi als a e
equi ed as a pe o mance s anda d o ha e epo ing sys ems in place o iden i y in-pa ien s wi h a
BMI  27 wi h a co-mo bidi y o BMI 30. We ha e audi ed compliance wi h his pe o mance
s anda d in wo hospi als. No es and clinical eco ds o all pa ien s we e inspec ed on sample
medical, su gical and acu e admission wa ds. Da a on heigh , weigh , BMI, wais ci cum e ence,
clinical s a emen abou body habi us, and cu en p esc ip ion o d ugs o ca dio ascula o
me abolic disease we e eco ded. Whe e heigh was no eco ded, an es ima ed BMI was calcula ed
using ‘a e age’ heigh o men (1.75m) and women (1.61m).
Table: Weigh , BMI and wais ci cum e ence eco ded on inpa ien s
An h opome y Hospi al An=156 Hospi al B
n=147
Weigh 110 (70.5%) 85 (57.82%)
Wais ci cum. none none
BMIs
eco ded 78 (50%) 14( 9.5%)
BMI ca ego y BMI30 BMI27 BMI<20 BMI 30 BMI 27 BMI<20
Reco ded 2415.3% 4126.2% 117.0% 96.1% 10(6.8%) -
Es ima ed 95.7% - 1610.9% 4127.9% 1510.2%
Unde - epo ing o BMI is common. Leng h o s ay impac ed upon weigh eco ds: weigh was
eco ded in no pa ien s wi h <2 days and only 80% wi h ≥8 days admission. Pa adoxically, hose
wi h CV o diabe es- ela ed condi ions we e less likely o ha e a weigh eco ded. Wais
ci cum e ence was no eco ded on any pa ien .
Recogni ion o o e weigh and obesi y (and also p esumably unde weigh ) emains poo in English
hospi als.
T5:PS.159
E ec s o exe cise on isk ac o s, exe cise capaci y and body composi ion in obese
indi iduals, class I-III.
Wisén A1, Sal in B2, Ch is iansen L1
1Bispebje gs Hospi al, Copenhagen, Denma k; 2Cen e o Muscle Resea ch, Rigshospi ale ,
Copenhagen Denma k
The e ec s o di e en exe cise in ensi ies in se e ely obese indi iduals (mean BMI= 42.1;
ange 33.3-64.8) we e explo ed. Subjec s we e andomised in o wo g oups and exe cise
pe o med o 16 weeks. Tes s we e pe o med p io and a e he exe cise pe iod.
Bo h g oups pe o med supe ised exe cise 3 imes a week. A high in ensi y g oup (HI,
n=51) exe cised o 1 hou , and a low in ensi y g oup (LO, n=43) o ½ hou , on each
occasion. The LO g oup also pe o med non-supe ised exe cise a leas 3 imes a week.
High in ensi y Low in ensi y
Tes 1 Tes 2 (n) Tes 1 Tes 2 (n)
Weigh (kg) 123 120 *** 51 122 120 * 43
BMI (kg ⋅ m2)42 40 *** 51 43 42 ** 43
Fa (% o body weigh ) 46.3 45.0*** 51 46.5 46.0 ns 43
Fa (kg) 54.5 51.6*** 51 55.0 53.5 ns 43
VO2max (L ⋅ min) 2.7 2.9 *** 51 2.2 2.3 * 43
VO2max (ml ⋅ min-1 ⋅ kg-1)22.3 24.8 ** 51 18.8 20.1 * 43
BPsys (mmHg) 153 144*** 28# 160 144** 24#
BPdias (mmHg) 99 90.8** 21# 99 90*** 20#
TG (mic oMol/L) 2104 1656* 26# 1955 1830ns 26#
LDL (mMol/L) 4.8 4.3* 20# 4.8 4.4ns 20#
Glucose (mMol/L) 8.2 7.2ns 13# 9.0 7.0** 9#
Table: Mean alues and s a is ical signi icance.
#Risk ac o s we e e alua ed, only o subjec s wi h ini ially inc eased le els.
In conclusion, all e alua ed pa ame e s we e nume ically imp o ed in bo h g oups (Table).
In he HI g oup, se e al pa ame e s imp o ed wi h g ea e signi icance han in he LO
g oup.
T5:PS.160
Changes o body weigh in ou doo pa ien s - ela ionship wi h mo bidi y and
medica ion du ing an obse a ion pe iod o 11 yea s
Dzien A1, Dzien- Bischinge C1, Hoppichle F2, Dämon S2, Lechlei ne M3
1Medical Cen e Innsb uck; 2KH Ba mhe zige B üde , Salzbu g; 3KH Hochzi l,
Innsb uck
Objec i e: In man
y
o e wei
g
h and obese pa ien s wei
g
h educ ion and wei
g
h
main enance a e di icul o ob ain, especiall
y
unde clinical ou ine p ocedu es. In
addi ion o counselin
g
p o
g
ams he dia
g
nosis o me abolic disease o ca dio ascula
diso de s could be o in luence on he pa ien `s mo i a ion o lose body weigh .
Me hods: Clinical and labo a o
y
esul s o 3193 pa ien s a endin
g
a medical
ou doo cen e we e e alua ed. Dia
g
nosis and he need o medica ion we e
compa ed be ween pa ien s wi h a success ul wei
g
h loss and hose wi h a con inuous
weigh gain du ing an obse a ion pe iod o 11 yea s.
Resul s: In he emale s udy popula ion (n=1844) he pe cen age o pa ien s wi h
no mal BMI was 46%, wi h o e wei
g
h 19%, wi h obesi
y
22% and wi h mo bid
obesi
y
2%. In he male s ud
y
popula ion
(
n=1349
)
44% o he pa ien s e ealed a
no mal BMI, 40% we e o e wei
g
h , 10% obese and 1% mo bidl
y
obese. A
counseling p og am comp isin
g
die a
y
ins uc ions and ecommenda ions o
physical ac i i y was o e ed o all o e weigh and obese pa ien s.
Du in
g
he ollowin
g
obse a ion pe iod o 11
y
ea s, success ul and main ained
weigh loss could be obse ed in 313 pa ien s wi h a mean BMI o 26.8±2.6 kg/m2a
he i s isi and 25.2±3.3 kg/m2
(
p<0.001
)
a he la es con ol. A he ini ial isi
his
g
oup o pa ien s wi h a success ul wei
g
h loss e ealed mo e un a ou able
me abolic esul s and a highe p e alence o ype 2 diabe es
(
12.5%
)
, h
y
pe ension
(
38%
)
and co ona
y
hea disease
(
19%
)
, han he
g
oup o 330 pa ien s wi h a
con inuous inc ease in BMI
(
y
pe 2 diabe es 7.2%, h
y
pe ension 25%, co ona
y
hea
disease 12%). Du ing he obse a ion pe iod o 11 yea s he p e alence o diabe es
inc eased o 1.2% in pa ien s wi h wei
g
h loss and o 1.8% in hose wi h wei
g
h
g
ain. The inc ease in ca dio ascula medica ion was 24% in pa ien s wi h wei
g
h loss
and 48% in he g oup wi h weigh gain.
Conclusion: The esul s o ou e alua ion indica e ha in addi ion o counselin
g
p
o
g
ams he dia
g
nosis o me abolic o ca dio ascula disease mi
g
h be a s on
g
mo i a o o pa ien s o lose bod
y
wei
g
h success ull
y
and o main ain his e ec o
yea s.