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Differential stimulus coupling to dopamine and norepinephrine stores in rabbit carotid body type I cells

Abstract

Recent studies suggest that preneural type I (glomus) cells in the arterial chemoreceptor tissue of the carotid body act as primary transducer elements which respond to natural stimuli (low 02, pH or increased CO2) by releasing chemical transmitter agents capable of exciting the closely apposed afferent nerve terminals. These type I cells contain multiple putative transmitters, but the identity of the natural excitatory agents remains an unresolved problem in carotid body physiology. Characterization of putative transmitter involvement in the response to natural and pharmacological stimuli has therefore become fundamental to further understanding of chemotransmission in this organ. The present study demonstrates that a natural stimulus (hypoxia) evokes the release of dopamine (DA) and norepinephrine (NE) in approximate proportion to their unequal stores in rabbit carotid body (DA release/NE release = 8.2). In contrast, nicotine (100/~M), a cholinornimetic agent thought to act on the nicotinic receptors present on the type I cells, evokes the preferential release of NE (DA release/NE release = 0.17). These findings suggest that distinct mechanisms are involved in a differential mobilization of these two cateeholamines from the rabbit carotid body.

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Differential stimulus coupling to dopamine and norepinephrine stores in rabbit carotid body type I cells

Author: Gómez Niño, María Ángeles,Dinger, Bruce,González, Constancio,Fidone, Salvatore
Publisher: Elsevier
Year: 1990
Source: https://uvadoc.uva.es/bitstream/10324/6849/1/Constancio%2013.pdf
160 B ain Resea ch, 525 (1990) 160-164 Else ie
BRES 24225
Di e en ial s imulus coupling o dopamine and no epineph ine s o es in
abbi ca o id body ype I cells
A. Gomez-Ni io, B. Dinge , C. Gonzalez and S.J. Fidone
Depa men o Physiology, Uni e si y o U ah School o Medicine, Sal Lake Ci y, UT 84108 (U.S.A.)
(Accep ed 1 May 1990)
Key wo ds: A e ial chemo ecep o ; Hypoxia; Nico inic ecep o ; Chemo ansmission; T ansmi e elease; Ca echolamine
Recen s udies sugges ha p eneu al ype I (glomus) cells in he a e ial chemo ecep o issue o he ca o id body ac as p ima y ansduce
elemen s which espond o na u al s imuli (low 02, pH o inc eased CO2) by eleasing chemical ansmi e agen s capable o exci ing he closely
apposed a e en ne e e minals. These ype I cells con ain mul iple pu a i e ansmi e s, bu he iden i y o he na u al exci a o y agen s
emains
an un esol ed p oblem in ca o id body physiology. Cha ac e iza ion o pu a i e ansmi e in ol emen in he esponse o na u al
and pha macological s imuli has he e o e become undamen al o u he unde s anding o chemo ansmission in his o gan. The p esen s udy
demons a es ha a na u al s imulus (hypoxia) e okes he elease o dopamine (DA) and no epineph ine (NE) in app oxima e p opo ion o
hei unequal s o es in abbi ca o id body (DA elease/NE elease = 8.2). In con as , nico ine (100/~M), a cholino nime ic agen hough
o ac on he nico inic ecep o s p esen on he ype I cells, e okes he p e e en ial elease o NE (DA elease/NE elease = 0.17). These
indings sugges ha dis inc mechanisms a e in ol ed in a di e en ial mobiliza ion o hese wo ca eeholamines om he abbi ca o id body.
The chemosenso y issue o he mammalian ca o id
body consis s o mo phologically dis inc ype I (glomus)
cells which lie in synap ic associa ion wi h a e en ne e
e minals o he ca o id sinus ne e (CSN), a b anch o
he glossopha yngeal (IX h) c anial ne e. A undamen-
al issue in ca o id body physiology conce ns he ole
played by ype I cells in chemo ansduc ion and chemo-
ansmission o na u al and pha macological s imuli (low
pO2, pH o ele a ed pCO2; and choline gic, pep ide gic
and ca echolamine gic agen s) 1~. Wi h espec o chemo-
ansduc ion, p e ious s udies ha e demons a ed ha
ype I cells espond o na u al s imuli in i o and in i o
in he absence o inne a ion 1°, hus implying ha hese
cells may be good candida es o he ole o chemosen-
so y ansduce in he ca o id body. Mo eo e , ecen
epo s ha e shown ha ype I cells possess a unique
O2-sensi i e po assium conduc ance, which is dec eased
by hypoxic s imuli wi hin he physiological ange 2~. Wi h
espec o chemo ansmission be ween ype I cells and
senso y ne e e minals, a majo p oblem emains how
he mul iple pu a i e neu o ansmi e s ound in ype I
cells (including biogenic amines and neu oac i e pep-
ides) a e able o in e ac in he ini ia ion and modula ion
o he chemosenso y discha ge o he CSN.
The pu a i e ansmi e s p esen in highes concen-
a ions in he ca o id body a e he ca echolamines
dopamine (DA) and no epineph ine (NE). While he
p ecise ole o DA emains unce ain, i is clea om
p e ious s udies wi h he abbi ca o id body ha he
syn hesis and elease o DA by ype I cells is inc eased
by na u al s imuli 1°'11, and ha dopamine gic (Dz)
ecep o s a e p esen on CSN a e en s 5'24. In con as ,
i ually no hing is known ega ding he egula ion o NE
me abolism in he o gan, and al hough i s syn he ic
enzyme (dopamine l-hyd oxylase; DBH) is known o be
p esen in ype I cells 2'27, a emp s o demons a e
NE/DBH sensi i i y o chemo ecep o s imuli ha e un il
e y ecen ly me wi h li le
success 14'15'26.
Likewise,
while he esul s o pha macological expe imen s sugges
ha ce ain chemo ecep o esponses by he ca o id body
may be media ed by l-ad ene gic ecep o s a2, a mecha-
nism coupling speci ic s imuli o NE u no e and
u iliza ion has ye o be demons a ed.
In he p esen s udy, we ha e examined he elease o
[3H]DA and [3H]NE (syn hesized om [3H] y osine)
e oked by wo classical chemoexci a o y agen s H (low 0 2
and nico ine) om abbi ca o id bodies supe used in
i o. While he da a indica e ha he elease o bo h
ca echolamines is inc eased by hese s imuli, i is clea
ha hypoxia e okes he elease o [3H]DA in p opo ion
o i s la ge con en in he issue, whe eas nico ine
p oduces a nea ly selec i e elease o [3H]NE. The esul s
he e o e sugges ha unc ionally dis inc mechanisms
egula e he u no e o endogenous DA and NE in he
Co espondence: S.J. Fidone, Depa men o Physiology, Uni e si y o U ah School o Medicine, 410 Chipe a Way, Resea ch Pa k, Sal Lake
Ci y, UT 84108, U.S.A.
0006-8993/90/$03.50 © 1990 Else ie Science Publishe s B.V. (Biomedical Di ision)
ca o id body. P elimina y epo s o hese indings ha e
appea ed elsewhe e 14'15.
Rabbi ca o id bodies we e i s ch onically sympa-
hec omized o elimina e he sympa he ic (NE-con-
aining) ne e e minals om he ca o id body. Unde
pen oba bi al anes hesia (35 mg/kg), supe io ce ical
sympa he ic ganglia we e bila e ally excised om adul
New Zealand whi e abbi s 10-14 days p io o expe i-
men a ion, a which ime he animals we e e-anes he-
ized and hei ca o id bodies su gically emo ed and
cleaned o adjoining connec i e issue in a ba h o
ice-cold modi ied Ty ode's solu ion (in mM: NaCI 112,
KCI 4.7, CaCI 2 2.2, MgCI 2 1.1, sodium glu ama e 42,
HEPES bu e 5, glucose 5.6, pH = 7.42) equilib a ed
wi h 100% 0 2 . The issues we e pooled and incuba ed o
2 h, as p e iously desc ibed 9, in media con aining
[3H] y osine (20-40 Ci/mmol; 20-25 / M; Ame sham)
plus asco bic acid (100/~M) and y osine hyd oxylase
co ac o (6-MPH4; 50/~M). Following incuba ion, ca o id
bodies we e sepa a ed in o g oups o 2-4 and washed o
90 min ( esh media e e y 30 min) in s anda d scin illa-
ion ials con aining modi ied Ty ode's solu ion equili-
b a ed wi h 100% 0 2. Ca echolamine (CA) elease was
de e mined u ilizing adiolabeling echniques in conjunc-
ion wi h HPLC sepa a ion o DA om NE, and was
assessed du ing s imulus cycles which each consis ed o
sequen ial con ol (100% O2-media ), s imulus (low 0 2-
o nico ine-con aining media) and pos -s imulus (100%
O2-media ) pe iods. Simila esul s o hose epo ed he e
we e ob ained when a bica bona e-CO 2 (95% 02/5%
CO2, pH = 7.42) bu e was used in place o he
HEPES-O 2 (100%
0 2 o
10%
02/90% N2)
bu e ; i.e.,
elease p o iles we e quali a i ely he same, and quan i-
a i ely wi hin 10% o each o he . Supe usa es we e
collec ed in ials con aining asco bic acid (10 mM), plus
ace ic acid (4 M), and s o ed o e nigh (4 °C). [3H]DA,
[3H]DOPAC and [3H]NE we e elu ed om alumina
columns (see e . 10) wi h 1 N pe chlo ic acid, and he
labeled p oduc s combined wi h unlabeled NE, DA and
DOPAC p io o HPLC sepa a ion on a C-18 e e se
phase column (Rainin) in phospha e-bu e ed mobile
phase. Sample aliquo s spiked wi h 3,4-dihyd oxyman-
delic acid (DOMA) and 3-me hoxy-4-hyd oxy-phenylgly-
col (MHPG) we e ch oma og aphed sepa a ely o quan-
i y hese NE me aboli es. CA was de ec ed elec ochem-
ically wi h an ESA Coulochem (Model 5100A; 5021
condi ioning cell; 5011 analy ical cell) in he educ i e
mode. Peaks o in e es we e collec ed au oma ically wi h
a Gilson Model 202 ac ion collec o o subsequen
coun ing in a Packa d Model 1500 scin illa ion spec om-
e e .
The da a p esen ed in Fig. 1 ( op) show o al [3H]CA
elease om an expe imen in which ca o id bodies we e
161
exposed o wo s imulus cycles; he i s cycle con ained
media equilib a ed wi h 10% 02, he second cycle
con ained 100% 02 equilib a ed media along wi h 100
/ M nico ine, which in p elimina y dose- esponse expe i-
men s had p o en o e oke an in e media e amoun o
[3H]CA elease. Be ween s imulus cycles he issue was
washed o 30 min wi h wo changes o 100% 02-
equilib ium media. We consis en ly obse ed a di e en
ime cou se o CA elease o low 0 2 s nico ine
s imula ion. CA elease e oked by low 0 2 pe sis ed in o
he i s pos -s imulus pe iod, whe eas nico ine-e oked
elease ended ab up ly a e he 5 min s imulus pe iod.
Re e sal o he s imulus o de esul ed in simila ela i e
pa e ns o CA elease by low 0 2 and nico ine. The
A
e*
0
a.
_e
9
20-
10-
0
////
~
Q
Hypoxia
I~
Nico ine
Time~
'0
0
m
m
0
m
9
I--
Hypoxia Nico ine
Collec ion Pe iods
(10 min. each )
I
Time
Fig. 1. Basal and s imulus-e oked [3H]ca echolamine ([3H]CA)
elease om 4 sympa hec omized abbi ca o id bodies supe used
in i o. The supe usion media we e eplaced a 10 min in e als,
excep du ing s imula ion (low 02 o nico ine) which las ed 5 min;
o compa ison pu poses, elease du ing hese pe iods was mul i-
plied by 2. Basal elease was e alua ed immedia ely p io o
s imula ion in media equilib a ed wi h 100% O 2. The ca o id bodies
we e s imula ed wi h media equilib a ed wi h 10% 02 o wi h media
equilib a ed wi h 100% 02 and con aining 100/~M nico ine, and
elease was e alua ed h ough 3 pos -s imulus pe iods. Be ween
s imulus cycles he issue was washed o 2 x 15 min in 100% 02
media. Uppe panel: o al
[3H]CA
eleased; lowe panel: p o ile o
3H-NE (open ba s), 3H-DOPAC
(c oss-ha ched ba s) and [3H]DA
( illed ba s) elease de e mined by HPLC and scin illa ion analysis.
162
easons o his di e ence in ime cou se o elease is no
known, bu ecep o desensi iza ion by nico ine 2°'22 mus
be conside ed a possible explana ion o u u e expe i-
men s add essing his issue. The ecep o speci ici y o
he nico ine e ec was es ablished in con ol expe imen s
which showed ha mecamylamide (100 ~M), hexame-
honium (100 k M) and (+)- ubocu a ine (50 ~M) blocked
mo e han 95% o he nico ine-e oked elease o
[3HICA.
Fig. 1 (bo om) shows he ela i e p opo ions o DA
and NE eleased du ing he wo s imulus cycles desc ibed
abo e. The con en o [3H]NE, [aH]DA and [SH]-
DOPAC a e exp essed as a pe cen o he o al [3H]CA
([3H]NE + [3H]DA + [3H]DOPAC) eleased in each
sample. In mos ins ances he NE me aboli es DOMA
and MHPG amoun ed o only 4-5% o he eleased
[3H]CA, and hey ne e exceeded 11% o he o al
[3H]CA elease ei he unde es ing condi ions o in
esponse o hypoxia o nico ine. I is appa en om he
da a ha he ela i e p opo ions o [3H]DA and [3H]NE
elease a e di e en o he low 0 2 s he nico ine
s imulus. Exposu e o low OE-media p ima ily e oked
elease o [3H]DA, while [3H]NE domina ed he p o ile
o nico ine-e oked elease. These di e ing pa e ns o
elease a e unlikely due o non-speci ic e ec s o he
s imuli, because exposu e o ca o id bodies o high K +
(75 aM) p oduced a elease p o ile which closely e lec s
he ela i e [3H]DA/[3H]NE con en in he o gan (no
shown).
Da a om mul iple elease expe imen s u ilizing low
0 2 and nico ine as sec e ogogues a e p esen ed in Table
I. [3H]CA elease in con ol (100% OE) supe usion
media du ing he 10 min pe iod immedia ely p io o
s imula ion consis ed o 16.9% [SH]NE and 83.1%
[3H]DA + [3H]DOPAC. Release du ing supe usion
wi h 10% OE-media was comp ised o 98.1% [aH]DA +
[3H]DOPAC. Al hough only a small po ion o he
e oked elease was due o [3H]NE, i s concen a ion in
he supe usion media was none heless app oxima ely
doubled by he hypoxic s imulus. Howe e , i mus be
conside ed ha he scin illa ion da a alone may no
accu a ely e lec he ela i e amoun s o NE and DA
eleased in hese expe imen s, because he speci ic
ac i i y o hese wo CA (and hei me aboli es) in he
issue may be di e en . Indeed, he speci ic ac i i y o
DA in he issue a e he expe imen was usually 2-5
imes g ea e han ha o NE, due o he slowe
u no e and syn hesis o NE by he ca o id body 9.
Con e sion o he ac ional adiolabelled con en e-
leased o he physiologically ele an pa ame e o
absolu e amoun eleased, equi es an es ima e o he
speci ic ac i i ies o he eleased [3H]NE and [3H]DA.
Expe imen s in ou labo a o y add essed his poin o
he elease o DA/[3H]DA e oked by hypoxia and
ele a ed K + (60 aM; e . 3). The esul s showed ha he
speci ic ac i i y o eleased [3H]DA (plus [3H]DOPAC)
was e y simila o ha o he [3H]DA emaining in he
issue. Thus, i appea s ha du ing he 90 min wash
pe iod be o e collec ion o samples, he newly syn he-
sized [3H]DA equilib a es wi h he endogenous unlabeled
DA pool in ou expe imen s. These esul s a e in
ag eemen wi h o he s udies desc ibing he inco po a-
ion o newly syn hesized NE in o he endogenous CA
pool in sympa he ic ne e e minals in he spleen 13'18.
Consequen ly, we used he speci ic ac i i y da a o
DA/[3H]DA and NE/[3H]NE con en om s imula ed
ca o id bodies in o de o calcula e he o al s imulus-
e oked elease o NE and DA, and hese esul s a e
p esen ed in Table II. The da a show ha con e sion
om dpm eleased/mg issue, o absolu e amoun e-
leased (pmoi/mg issue) con i ms he p edominance o
DA elease by hypoxia and ac ually exagge a es he
p edominance o NE elease by nico ine. The absolu e
amoun s o NE and DA eleased in esponse o hypoxia
we e app oxima ely in p opo ion o he ela i e con en
o hese wo CA in he issue, whe eas in esponse o
nico ine he nea ly 5- old p e e ence o NE elease
e lec s a ma kedly disp opo iona e mobiliza ion o NE
s DA s o es in hese sympa hec omized ca o id bodies.
The da a addi ionally show ha basal elease consis ed o
TABLE I
Release o adiolabeled CA om sympa hec omized abbi ca o id bodies measu ed in media equilib a ed wi h 100% 02 (con ol), 10% 02 o 100%
02-media con aining lO01~m nico ine
Values a e means + S.E.M. wi h numbe s o samples in b acke s.
dpm/mg issue
[3H]NE
% o o al CA
[3 I]DOPA C + [3H]DA [3HINE [3H]DOPAC + [3H]DA
Con ol(basal) 242 + 37 (16) 1193 _+ 191" (16)
10% 0 2 538 +- 77* (11) 28138 -+ 7946* (11)
100/~M nico ine 4223 + 78* (6) 2747 _+ 410" (6)
*P < 0.001 s basal elease.
16.9% - 2.6% 83.1% + 13.3%
1.9% -+- 0.3% 98.1% --- 27.7%
60.1% + 1.1% 39.9% -+ 5.9%
163
TABLE II
Absolu e amoun s o CA eleased (pmol/mg issue) calcula ed om measu emen s o he speci ic ac i i y o DA/[3H]DA and NE/[3H]NE in
s imula ed ca o id bodies
Values a e means + S.E.M. wi h numbe s o samples in b acke s.
pmol/mg issue % o o al CA
NE DOPAC + [3H]DA NE DOPAC + DA
Con ol (basal) 0.227 + 0.03 (16) 0.224 + 0.04 (16) 50.3% + 6.7% 49.7% + 8.9%
10% 02 0.833 + 0.16" (11) 6.851 + 1.936" (11) 10.8% + 2.1% 89.2% + 25.2%
100/~M 3.143 + 0.49* (6) 0.544 + 0.09* (6) 85.2% + 13.3% 14.8% + 2.4%
*P ~< 0.001 s basal elease.
app oxima ely equal po ions o DA and NE.
Ou indings demons a e ha nico ine and hypoxia
di e en ially mobilize NE s DA om ca o id body CA
s o es. Howe e , he e is no comple e selec i i y by
hese s imuli, because hypoxia consis en ly e oked he
elease o small amoun s o [3H]NE, while [3H]DA was
eleased a ela i ely low le els in esponse o nico ine
(see Table I). In his ega d, i may be impo an o
conside ha elease-p omo ing e en s migh be occu -
ing seconda y o he ini ial s imulus-induced elease; o
example, he ac i a ion o l-ad ene gic ecep o s in he
ca o id body has been epo ed o e oke DA ou low 25,
and consequen ly a possible sequence o e en s migh
in ol e i s a selec i e nico ine-induced elease o NE,
ollowed by l- ecep o ac i a ion and he subsequen
elease o DA. In addi ion, p elimina y expe imen s
which examined he e ec s o combined s imula ion wi h
nico ine and hypoxia showed ha he o al [3H]CA
elease was la ge han he sum o he elease e oked by
each condi ion, sugges ing in e es ing in e ac i e e ec s
o hese s imuli.
The obse a ion ha hypoxia and nico ine e oke he
elease o DA and NE om he ca o id body in dis inc ly
di e en p opo ions sugges s ha hese s imuli ac i a e
di e se mechanisms o mobilizing hese wo CA om
he ype I cells o his chemosenso y o gan. Al hough i
has been conjec u ed ha sepa a e dopamine gic and
no ad ene gic cells may exis in he ca o id body 1,
2,4,16,17.23, ecen immunocy ochemical s udies ha e
shown ha i ually all ype I cells con ain bo h y osine
hyd oxylase (TH), he a e limi ing enzyme o CA
syn hesis, and dopamine- l-hyd oxylase (D lH), he syn-
he ic enzyme o NE p oduc ion 2'27. While hese s udies
sugges a ubiqui ous dis ibu ion o DA and NE amongs
ype I cells, he le els o immunocy ochemical eac ion
p oduc o D lH epo ed in hese s udies indica e ha
some cells may con ain much mo e NE han o he s. This
possibili y, coupled wi h he obse a ion by Chen and
Ya es I ha speci ic a-bunga o oxin binding si es in he
ca o id body a e es ic ed o a subg oup o ype I cells
(dis inguished on he basis o dense-co e esicle size;
e s. 16, 23), sugges s he possibili y ha he obse ed
p e e en ial elease o NE o e DA migh a ise om he
selec i e dis ibu ion o nico inic ecep o s on cells which
con ain ela i ely high le els o NE. Al e na i ely, ype
I cells in he ca o id body may ep esen a homogeneous
popula ion wi h espec o hei con en o DA and NE
and hei sensi i i y o nico ine. Di e en p opo ions o
CA elease would hen be achie ed h ough he selec i e
mobiliza ion o dense-co ed esicles con aining ei he
mos ly DA o NE. The a ailable da a a e equi ocal wi h
espec o he cellula mechanisms in ol ed in he
obse ed di e en ial elease o CA, and al hough ecen
immunocy ochemical s udies o TH and D lH ha e
p o ided use ul in o ma ion in his ega d, i mus also be
ecognized ha he demons a ed p esence o a ans-
mi e 's syn he ic enzymes do no necessa ily signal he
p esence o he ansmi e 19.
Finally, he in ol emen o nico inic ecep o s in he
esponse o ype I cells o chemo ecep o s imuli (na u al
and pha macological) sugges s a ole o ACh in chemo-
sensa ion. Mo e han 20 yea s ago, Eyzagui e and
Zapa a s demons a ed ha anoxia o elec ical s imula-
ion o he ca o id body e oked he elease o an
ACh-like subs ance om he o gan. Ea ly neu ochemical
and au o adiog aphic s udies placed ACh in he ype I
cells 1~, and his p ospec was i mly es ablished by ecen
immunocy ochemical s udies localizing o hese cells he
syn he ic enzyme o ACh, choline ace yl ans e ase
(CHAT) 28. Ligand binding s udies using a-bunga o oxin
and quinuclidinylbenzyla e (QNB) ha e u he demon-
s a ed ha choline gic ecep o s a e loca ed on ype I
cells 6'7. The cu en demons a ion ha nico inic ecep-
o s e oke he p e e en ial elease o NE om he ca o id
body emphasizes he need o u he expe imen a ion
ega ding he physiological oles played by ACh and NE
in he ca o id body.
Suppo ed by USPHS G an s NS12636 and NS07938, and
DGICYT PB86-0325 (Spain).
164
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13 (1984) 281-302.
2 Chen, I.L., Hansen, J.T. and Ya es, R.D., Dopamine l-
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body: a ligh and elec on mic oscopic s udy,
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14
(1985) 131-144.
3 Cheng, G.-E, Dinge , B. and Fidone, S.J., The elease o
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13 (1987) 1111.
4 Chis ie, D.S. and Hansen, J.T., Cy ochemical e idence o he
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